The Effects of Fertilizer on Plant Growth Research Paper

The Effects of Fertilizer on Plant Growth - Research Paper Example â€Å"A recent assessment found that about 40 to 60% of crop yields are attributable to commercial fertilizer use† (Coleman, Fuenta, and Mock 1978). There are many types of fertilizer out there today for the aid of plant growth. There are many ways to categorize types of fertilizers; they can be classified as either organic or inorganic. It can also be defined as a solid or a liquid fertilizer, along with different ingredients that produce different actions. â€Å"Organic fertilizer is all natural and includes things such as bat guano, compost, peat moss, wood ash and manure. These are general soil amendments. They don't burn or harm plants, and they can have long-term positive effects on the soil without damaging groundwater. Organic fertilizer, however, generally has lower nutrient concentrations than inorganic fertilizers† (Broschat and Moore 2003). Whereas inorganic fertilizer can be classified with the characteristics of. â€Å"Man-made and typically comes as a po wder, pellets, granules or a liquid. Other chemicals that might be included in inorganic fertilizers include calcium, sulfur, iron, zinc and magnesium† (Broschat and Moore 2003). Research Questions 1. What are the specific effects of fertilizer on plant growth? 2. ... Methods Soil was mixed using a 1:1:1 ratio of pet moss, vermiculite, and potting soil. One scoop of the soil was collected and misted with water until it was moist. Four potting trays with six cells each were gathered. The cells were filled half way with the moist soil. Five osmocote pellets were added to each of the cells. The cells were completely filled with the remaining moist soil. A pencil was used to poke three tiny holes in each of the cells. A seed was placed at the top of each hole. The holes were then lightly covered with soil. Each of the cells was labeled one through twelve for the control group and the experimental group. A diamond wick was inserted into each of the cells halfway and was folded over at the bottom. This was to help water enter the cell. Two tubs were filled with water and two pieces of fabric for each tub were soaked and draped over the top of the boxes. Two antiagal tabs were added to each tub of water to prevent algae from growing. One control tray and one experimental tray were placed on each of the tubs. The trays were mixed up so that if something happened to one of the tubs, there would still be a tub that could be examined. The trays were placed under the fluorescent bulbs in the classroom. Plant height, number of leaves, and number of buds were collected each week. Water was added to the tubs weekly as well. Data was analyzed using statistical t-tests. Ethical Considerations Ethics are the moral codes which are followed in a research. These codes are binding and need to be followed irrespective of any circumstance which may surround the research since they give us a remembrance of the researcher’s responsibility towards the people being researched (Chapman and Shaw 2000). The following

Taming of the Shrew Character Motivations Essay Example for Free

Taming of the Shrew Character Motivations Essay Many lives are ruled by a need to fit in and conform to the ideals of society. William Shakespeare’s Taming of the Shrew centers around the two sisters, Bianca and Katherine and their marriages. Kate, an unpleasant and undesirable woman, unwillingly marries Petruchio who attempts to tame her. Bianca is the ideal woman who is pursued by many men. The play was written in 1590 and set in Italy, when submission and obedience were expected of women while men took powerful positions. Societal expectations motivate men and women. Women were motivated by societal expectations. A valued woman was obedient, passive and sweet. Bianca epitomizes these qualities and shapes her behavior around them. She exchanges vicious words with her sister in private, but while in the company of suitors she turns mild. Bianca’s father and her suitors determine who she marries, showing her surrender to men’s power and acceptance of obedience. In Kate’s concluding speech, she says, â€Å"A woman moved is like a fountain troubled, muddy, ill-seeming, thick, bereft of beauty, and while it is so, none†¦will deign to sip or touch one drop of it. (5. 2. 151) Kate admits that an unpleasant, angry wife is undesirable and useless. Certainly no man will consider an ugly, rude woman worthy of his company. She finally relents to Petruchio’s taming and takes a woman’s normal place in the social hierarchy. Women conform to society’s strict expectations. The expectation that men are powerful and all-knowing influences men’s behavior. A m an’s masculinity and power was dependent on his control over his wife. Petruchio, Hortensio and Lucentio make a bet, â€Å"And he whose wife is most obedient†¦shall win the wager. † (5. 2. 15) This shows how much men value an obedient wife, which demonstrates their power. Each man hopes that he wins because a powerful man is valued. Baptista, the father of Kate and Bianca, arranges his daughters’ marriages and marries Kate off even though she is unwilling. Society’s standards permit this destructive and inconsiderate behavior. In their patriarchal society, women can’t show much preference in their suitors. Men are expected to make the best decision, and Baptista goes along with this standard. Wife taming was a common idea in this time and many men attempted it. Petruchio tries to tame Kate by starving, embarrassing, overruling and insulting her. Kate comments on the sun, but Petruchio contradicts her and says â€Å"Now, by my mother’s son, and that’s myself, it shall be the moon or star or what I list. †(4. 5. 6) Petruchio says anything he says is correct, enforcing the idea that women should surrender to a man’s authority. Petruchio tames Kate because of the social belief that men should be more powerful than their wives, therefore social expectations motivate him. Society’s standards motivate men’s behavior. Men and women modify their behavior to meet the ideals of society. Katherine transforms herself into a submissive wife and Bianca maintains her behavior as an obedient, charming woman. Petruchio tames Kate to show his masculinity and Baptista controls his daughters lives without regard for their feelings. In conclusion, societal expectations motivate characters in The Taming of the Shrew.

Dissection of Microrna-30Ds Function Roles

Dissection of Microrna-30Ds Function Roles DISSECTION OF MICRORNA-30DS FUNCTION ROLES IN MAMMALIAN PANCREATIC-BETA CELLS By:  Yiping Mao   ABSTRACT MicroRNAs (miRNAs) are a group of small non-coding RNAs (about 21-22 nucleotides long) that fine tune target protein output through messenger RNA degradation or inhibition of its translation. Recent studies showing that miRNAs and their function components respond to cellular stress to maintain steady state physiology of the cells. Since there are thousands of microRNAs existing in all kind of cells, their functional characterization during the normal states or stress conditions is not fully addressed yet. In this thesis, important aspects of pancreatic ÃŽÂ ²-cell function under normal or stress condition such as apoptosis, proliferation, insulin production and release and their regulation by the miRNA were explored. Pancreatic ÃŽÂ ²-cell is a group of insulin producing cells and plays critical role in maintaining glucose hemostasis. By combining mouse and cell line genetic approaches, high-throughput deep sequencing, a list of cell assays and molecular techniques, we have shed light on the novel roles of miR-30d, one highly expressed miRNA when ÃŽÂ ²-cell responding to high glucose stimulus, in regulating ÃŽÂ ²-cell mass on the middle aged mice. We demonstrated that overexpress of miR-30d deteriorated glucose tolerance ability of the mice with or without high fat diet treatment by significantly reducing the ÃŽÂ ²-cell mass with less insulin production. Additionally, we demonstrated that the reduced ÃŽÂ ²-cell mass is because both apoptosis pathway and proliferation p athway have been effected by miR-30d by targeting variety of protein factors expression. BCL2 interacting protein 3 (BNIP3) and cyclin E2 (CCNE2) have been respectively confirmed as miR-30ds targets and the effect of their regulation by miR-30d in pancreatic ÃŽÂ ²-cell proliferation or apoptosis aspects have been addressed as well. Furthermore, we could show that silencing of miR-30d in MIN6 cell (ÃŽÂ ²-cell mimicking cell line) by CRISPR-CAS9 gene editing system promotes the insulin secretion, which is through potentiated expression of MAFA, an insulin transcription factor. These studies uncovered novel functional roles of the miR-30d pathway in mediating ÃŽÂ ²-cell function and fate. Further dissection of these pathways shall uncover several mechanisms by which the ÃŽÂ ²-cells undertake to maximize their efficiency during disease states such as T2D.   CHAPTER 1: INTRODUCTION AND BACKGROUND 1.1 The Brief History of Diabetes and Research Long before terming diabetes, which means to pass through by Greek Apollonius of Memphis in 250 BCE and the re-discovery of honey-like-urine (glycosuria) by Thomas Willis, who included the term mellitus, Diabetes has discovered its first saying around 1500 B.C. in an Egyptian manuscript. It was perceived as a disease related with too great emptying of the urine (polyuria). Later, important discoveries includes Matthew Dobsons first proof of elevated urine and blood glucose levels (hyperglycemia) in people with diabetes (Dobson, 1776). In 1889, Joseph von Mering and Oskar Minkowski were the first to give evidence that pancreas removal in dogs induced diabetes, proposing that pancreas functional related to glucose levels. Afterward, Edward Albert Sharpey-Schafer proposed that diabetes could be brought on by losing a pancreatic chemical, which he named as insulin in 1910 (Polonsky, 2012). Taken together, diabetes is currently well recognized as a gathering of heterogeneous disorders cha racterized by hyperglycemia because of loss of insulin or its effectiveness. Current worldwide trends demonstrate a surprising 382 million individuals have diabetes and this is predicted to ascend to 592 million by the year 2035 (IDF Diabetes Atlas, 2013). The cases of diabetes complications, including diabetic retinopathy, cardiovascular disease and renal failure are constantly rising and the death rate because of those are worsen every year. Claude Bernards identification of liver as the major glucose production organ led to the first concept of homeostasis which has been termed and expanded by Walter Bradford in the mid-nineteenth century, to describe the maintaining of steady-state physiology of the cells (Robin, 1979). This gave the notion that actually the disturbed glucose homeostasis is one of the important events of the diabetes progression. Given the evidences of insulin is involved in maintaining the glucose homeostasis, Frederick Banting and Charles Best integrated a series of scientific approaches, and were able to purify the insulin from the pancreas. Moreover, they successfully treated the patients who suffer from the diabetes, with their purified insulin (Banting Best, 1922; Banting et al., 1922). This landmark finding set the stage for treating the severe diabetes with insulin. However, it has been almost a century now since the first time insulin was discovered and purified, diabetes remains the incurabl e disease, requires life-time attention and treatment because of its complexity. Diabetes has been classified as a couple different types nowadays, the major two types are known as the Type I Diabetes (T1D) or Insulin Dependent Diabetes Mellitus (IDDM) and Type II Diabetes (T2D) or Non-Insulin Dependent Diabetes Mellitus (NIDDM). Surprisingly, these two major types has been notified as early as 100-200 B.C by Indian physicians (Kahn, 1994). However, the first scientific evidence was brought by Wilhelm Falta and Harold Himsworth after they set the standardized glucose/insulin tolerance test in human to distinguish the insulin sensitive from non-sensitive patients (Himsworth, 1936). Insulin dependent T1D is featured in insufficient insulin production due to autoimmune response to the pancreatic beta-cells and it mostly affects young kids. T2D is more described as a metabolic syndrome with emergence of insulin resistance and obesity etc, and it is more common among adults (Moller, 2001). There is a complex network of several insulin responding tissues contributes to the maintenance of glucose homeostasis, and of course any abnormality in this network induces the progression of T2D. Besides the metabolic relevant tissues, the gene-interactions also play critical roles in the development of T2D and obesity such kind of metabolic diseases (Doria et al., 2008). 1.2 The Islet Architecture, ÃŽÂ ²-cell Fate determination The islets of Langerhans are specialized endocrine part of pancreas and are the only part producing the secreting hormones. They basically comprise of various cell sorts named ÃŽÂ ±, ÃŽÂ ², ÃŽÂ ´, PP, and ÃŽÂ µ that secret the islet hormones glucagon, insulin, somatostatin, polypeptide Y, and ghrelin individually, and these hormones are required to maintain the glucose homeostasis at normal or stress state. Furthermore, the islets are known to have dynamic and plastic architecture that is proposed to be adjusted over the time of development (Steiner et al., 2010). During the progression of insulin resistance, pregnancy or T2D, the islets extend in size to make up for expanded insulin requirement (Weir and Bonner-Weir, 2004; Kim et al., 2009). But afterwards, there is a considerable loss of ÃŽÂ ²-cell mass because of environmental or genetic factors, inducing the serious hyperglycemia in cause of insulin deficiency. While islets count to just ~1-2% of the whole pancreas, the insulin producing ÃŽÂ ²-cells represent ~65-80% of the islet mass, constituting to roughly 2% of pancreatic weight. Furthermore, the rest other cell types are considered as non ÃŽÂ ²-cells of the islets (Weir and Bonner-Weir, 2013). During the development, these distinctive cell types are known to emerge from a single progenitor cell that producing Neurogenin3 or Ngn3, a transcription factor that decides endocrine cell destiny (Edlund, 2002). Afterwards, other transcription factors, for example, Pdx1, Pax4, Nkx2.2, Nkx6.1, MafA, and Foxo1 help the ÃŽÂ ²-cell fate determination (Ziv et al., 2013). While it had been learned as that ÃŽÂ ²-cells proliferate by self-duplication from old ÃŽÂ ²-cells instead of differentiation from stem cell (Dor et al., 2004), some other study suggested that multipotent cells inside the pancreas could differentiate into ÃŽÂ ²-cells as well (Xu et al., 2008). The latter study is a lso confirmed by researches showing how expressing the ÃŽÂ ²-cell specific transcription factors in non ÃŽÂ ²-cell could trigger a ÃŽÂ ²-cell lineage in mice (Collombat et al., 2009; Al-Hasani et al., 2013). Recent studies additionally showing that non-ÃŽÂ ² cells, for example, ÃŽÂ ±-and ÃŽÂ ´-cells could experience transdifferentiation into ÃŽÂ ²-like cells when the mice is suffering from significant ÃŽÂ ²-cell loss (Thorel et al., 2010; Chera et al., 2014). On the other hand, a few researches demonstrate that the ÃŽÂ ²-cells can likewise lose their fate or dedifferentiate into non-ÃŽÂ ² or progenitor cells when losing any of the previously mentioned ÃŽÂ ²-cell particular transcription factor (Ziv et al., 2013). Moreover, a current study demonstrated that human ÃŽÂ ²-cells are capable of converting into ÃŽÂ ±-cells with no genetic modification (Spijker et al., 2013). Taken together, all these researches have showed that the dynamic plasticity of islet cells. 1.3 Glucose Stimulate Insulin Secretion One unique and significant feature of ÃŽÂ ²-cells is to detect the blood glucose changes and secrete insulin into extracellular milieu in response to keeping the glucose levels within the range of 4-8mM (Weir and Bonner-Weir, 2013). This is primarily accomplished by the take-up of extracellular glucose by the glucose transporter 2 (Glut2; Bell et al., 1990) at the plasma membrane. Upon uptake, the intracellular glucose sensor, glucokinase (Gck), subjects glucose moieties to quick metabolism system by glycolysis (Matschinsky and Ellerman, 1968). This brings about the producing of three carbon products: pyruvate, which takes part in the tricarboxylic corrosive (TCA) cycle inside the mitochondrion to eventually create adenosine triphosphates (ATP) by means of the electron transport chain system. The ATP therefore leads to the increase of ATP/ADP proportion in the cytoplasm, activating the closure of the ATP sensitive potassium (KATP) channel. Vitally, mutation in the kir6.2 subunit o f this channel were demonstrated to induce neonatal diabetes in both mice and human because of loss of insulin secretion as a consequence of constitutively open KATP channel (Koster et al., 2000; Gloyn et al., 2004). It has long been realized that glucose stimulates the closure of these KATP channels thus leading the slow menbrane depolarization (Ashcroft et al., 1984). This promotes extracellular calcium influx by voltage dependent calcium channels and potentiates the releasing of insulin (Matschinsky et al., 1998). Insulin is secreted in an oscillatory manner because of the blood glucose level and triggers downstream insulin signaling cascade in insulin-responsive tissues for the taking up glucose. It has well been shown that islets can be entrained to small changes in glucose and thus the plasma insulin has high frequency of oscillation. However, this capacity of entrainment of the islets is disturbed in patients with T2D (Mao et al., 1999). It exhibits ÃŽÂ ²-cell malfunction because of loss of insulin secretion is a major issue during the clinical indication of T2D. 1.4 Compensatory Islet Expansion During Insulin Resistance During the state of insulin resistance or over-weight, elevated plasma insulin levels (named hyperinsulinemia) has been found in polygenic mouse models showing insulin resistance and human subjects because of increasing of insulin secretion (Yalow and Berson, 1960; Polonsky et al., 1988; Brà ¼ning et al., 1997). It has been suggested later that both in rodents and people, this improved insulin secretion is apparently because of an expansion in ÃŽÂ ²-cell mass by either ÃŽÂ ²-cell proliferation (Steil et al., 2001) or ÃŽÂ ²-cell hypertrophy (Weir and Bonner-Weir, 2004). On the other hand, ÃŽÂ ²-cell failure because of different genetic or environmental variables, is known to cause declined plasma insulin levels in diabetics (Maclean and Ogilvie, 1955). It has been showed that lessened levels of insulin are frequently associated with a noteworthy loss of ÃŽÂ ²-cell mass because of ÃŽÂ ²-cell apoptosis (Butler et al., 2003; Rhodes, 2005). Other than the diabetes perspective, it has been demonstrated that matured ÃŽÂ ²-cells have long life-span and low proliferative rates at steady state. This is because of a potential limitation of the entry of matured ÃŽÂ ²-cells into cell cycle (Teta et al., 2005; Kushner, 2013). Other than this perception, later study suggested that adult ÃŽÂ ²-cells do have the ability to proliferate (Stolovich-Rain et al., 2012). In light of these findings on ÃŽÂ ²-cell proliferation, a few research groups have revealed various proteins essential for assisting ÃŽÂ ²-cell proliferation on knockout or transgenic mouse. Known cell cycle controllers including Cyclins D1 and D2, Cyclin subordinate kinase 4 (Cdk4), Cdk inhibitors (CKIs) such as Cip/Kip and INK4, transcription factors Retinoblastoma (Rb) and p53, have been proved on genetic mouse models as regulators of ÃŽÂ ²-cell proliferation and survival (Heit et al., 2006). Although transient high glucose has been considered as the result of insufficient insulin secretion or insulin resistance, it has also been revealed to promote the compensate ÃŽÂ ²-cell mass expansion (Bonner-Weir et al., 1989). This hypothesis was further supported recently by another observation, that it is the glucose metabolism, instead the glucose itself that triggers compensatory ÃŽÂ ²-cell proliferation in vivo (Porat et al., 2011). Some other attentions have been centered on the effect of activation of insulin/IRS2 pathway on driving ÃŽÂ ²-cell proliferation. The components of the pathway including IRS2 (Withers et al., 1998) and AKT (Bernal-Mizrachi et al., 2001) were demonstrated to be fundamental for ÃŽÂ ²-cell survival. Moreover, study has shown that the impact of insulin in ÃŽÂ ²-cell proliferation is even stronger when with hyperglycemia (Paris et al., 2003). It is notable that when in the state of severe insulin resistance, the pancreatic islets adjust t hemselves to meet the expanding requirement for producing and secretion more insulin by increasing their ÃŽÂ ²-cell mass (Weir and Bonner-Weir, 2004). Other than the involvement of protein coding genes, a few non-coding RNAs (ncRNAs), mostly microRNAs and long ncRNAs (lncRNAs), have been shown in the diabetogenes list, adding on to the complex genetic architecture of human diabetes. 2.1 The Brief History of MicroRNAs The most recent decade has seen huge attention regarding a new and special class of little ncRNAs such as microRNAs (miRNAs) in regulating the structure and function of ÃŽÂ ²-cells. With the first observation of a miRNA, lin-4 in C. elegans, researchers showed how a gene product encodes two little RNAs, instead of a protein. Besides, they demonstrated that these small RNAs binds to the compensatory sites at the 3 end of untranslated region (UTR) of lin-14, a development related heterochronic gene. This interaction is appeared to negatively regulate the expression of lin-14 by blocking its translation (Lee et al., 1993; Wightman et al., 1993), proposing miRNAs as negative regulators of gene expression. Even since the discovery of microRNAs, a lot of related research results have updated the mechanism of gene regulation to a novel level. miRNAs now are known as a group of small ncRNAs of ~21-22 nucleotides long that can complementary or non-complementary base-pairing the mRNA of protein coding genes, thus to regulate their expression at post-transcriptional level (Bartel, 2004 and 2009). Actually after the identification of lin-4, another miRNA called let-7 was revealed like lin-4 in both biogenesis and function level. Soon after, there starts a prevail in discovering new microRNASs mostly by high throughput sequencing technologies in research area and surprisingly, about 30,000 miRNA over about 200 species have been identified, which includes about 2,500 mature human miRNAs. (Kozomara and Griffiths-Jones, 2014). Many computational methods have also been produced to predict the potential targets of miRNAs based on the stable miRNA-target mRNA binding model (Lewis et al., 2003; Krek et al., 2005). 2.2 MicroRNA Induced Gene Silencing The intercellular gene silencing mechanism, termed as RNA silencing (RNAi) or post-transcriptional gene silencing (PTGS) is currently well known to be led by a group of small RNAs, for example, short interfering RNA (siRNA), piwi interacting RNA (piRNA), or the miRNAs. Basically, their working mechanisms are similar and the difference exist mostly in their biogenesis inside the cells (Ender and Meister, 2010). Mature miRNA producing has been through several steps: transcribed from DNA by RNA polymerase II, primary miRNA (pri-miRNA) has much longer sequence. Once transcribed, the pri-miRNA is further processed to precursor miRNA (pre-miRNA) about 60 nucleotides long by enzyme Dorsa and DGCR8 protein complex (Lee et al., 2003). Once pre-miRNAs are produced inside the nucleus, it will be export out of the nucleus by protein Exportin 5 to the cytoplasm in a Ran-GTP dependent manner (Lund et al., 2004). Another important enzyme, which is also critical for mouse development, Dicer would re cognize the pre-miRNA and process it to about 22 nucleotides long mature miRNA duplex form (Bernstein et al.,2003). Only one of stands of the duplex will be transported to miRNA-induced silencing complex (RISC) by Dicer, and the other strand, termed miRNA* is usually degraded in the end (Schwarz et al., 2003; Ender Meister, 2010). There are several components on the RISC, and one key protein component is AGO family. There are four well characterized members of AGO family in human AGO1, AGO2, AGO3 and AGO4. And AGO2 is expressed more often than other forms (Su et al., 2009; Wang et al., 2012). All the AGO proteins have the ability to slice mRNA because of their PAZ and PIWI cleavage domains under the guidance of miRNA sequence. Each miRNA has an important seed region, typically from 2nd-7th nucleotides, that could fully or partially bind to the mRNA 3 UTR sequence. The base pairing condition between the miRNA and target mRNA determines the target recognition and binding of miRISC, but also the fate of the mRNA- to be cleaved or to be repressed in translation. In animal system, the miRNA does not fully complementary bind to the 3 UTR of the target mRNA, through blocking the translation machinery, miRNA silencing the gene expression, without interference of the target stability. However, recent studies on the miR NA mediated gene silencing in mammalian cells reveals that miRNA may act through two step modes: at first, repressing the translation, then deadenylation and destabilization of target mRNA [27qualifier]. The deadenylation has been suggested to induce mRNA degradation [48] and translation inhibition is probably the requisite of mRNA degradation in mammalian cells [49].

Choose to scenes from the film that illuminates the text of Educating :: English Literature

Choose to scenes from the film that illuminates the text of Educating Rita by Willy Russell When the play was written there was a still a cultural divide between the uneducated (as in no higher education) working class and the cultural and educated upper class but higher education was slowly being madeavailable to the masses. This was partly due to Open University (OU) with provided a university education to those who normally would have not been able to go due to employment or the fact that they might feel they wouldn't fit in, like Rita, because of the cultural differences. Rita feels she needs an education to fill her life, as she knows that something in her life is not right and so she turns to the part of a life she doesn't have -education and knowledge of culture- to fill the gap. However as we find out this is not necessarily what she is missing, but in her search for an education she finds 'a new Rita', she even changes her name back to Susan. Susan is Rita's real name, however when she started OU she changed it to Rita after Rita Mae Brown, someone who she thinks writes literature, but in fact writes "pulp- fiction" stories, because at this time she thinks all books are literature- this shows the cultural difference. The play Educating Rita by Willy Russell gained great popularity especially during the early eighties. There has also been a movie made from it starring Julie Walters and the more famous Michael Caine. As so often the case, the movie was more elaborate with additional scenes, some of which were spoken of or retold by the actors in the play. The movie also included several actors while the play only featured two, Frank and Rita. In this essay I am going to explore how the film illuminates parts of the text when performed on stage. Although most of the details are small and subtle, they have a great impact on how the story portrayed. The movie offers much more background information on other characters and events that are important to the story. The play is much more restricted in the sense that a great many things are bound not to happen on stage, especially as there are only two characters. In fact nothing taking place outside Frank's office can be seen by the audience. All action is then confined within the stages four walls. When Frank invites Rita to his home for dinner in the play the

Reference Letter

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Fallacies of Irrational Thinking essays

Fallacies of Irrational Thinking essays The Fallacies of Irrational Thinking is the topic of this paper. To understand this concept you must first know the definition of the term fallacy. Fallacy is defined as deception, an error in logic, or an often plausible argument using false or invalid inference. In this course we learned that there were eight fallacies: perfection, approval, should, overgeneralization, causation, helplessness, and catastrophic causation. In this paper I will attempt to explain each of them. The fallacy of perfection is when a person that thinks that everything must be perfect. It can be as innocent as someone who must keep their things neat and clean or as extreme as a person cleans constantly because they don't feel that anything is clean enough. In their eyes, everything must be flawless. A person with this fallacy may most commonly be known as a perfectionist. One of their setbacks is that they are some busy perfecting that they don't have time to do other things. The fallacy of approval is when a person that insures that they say and do is acceptable to others. They are people pleasers, willing to do anything to make everyone happy and often say what the listeners want to hear. They are sometimes known as two-faded because they are on everyone's side of an argument. Most often affected by this fallacy are adolescents because they thrive on peer approval. A downfall is that the individual is so consumed with pleasing other the it may be difficult for them to The fallacy of should is when a person confuses what they want with what should be. They never understand why things are not going their way. This fallacy, unlike most of the others, can sometimes be beneficial, in moderation, in careers fields such as law enforcement and politics. In these field, in general, what the individual want usually will benefit public. On the other in hand, an extreme fallacy of should can be disasterist. One such exampl ...